Human Biochemistry, Volume 2 - Murray R. 1993

Special Topics
Nutrition, Digestion, and Absorption
Mineral Requirements

Minerals essential for physiological Functions can be broadly divided into two groups: 1) macrominerals, which are required in amounts exceeding 100 mg per day, and 2) Trace Elements (microminerals), whose daily requirement does not exceed 100 mg. The relevant data are presented in Table 53.1. Table 53.8 summarizes The properties of macrominerals, and Table 53.9 outlines the properties of trace elements.

Class="center">Table 53.6. Major Fat-soluble Vitamins. Principal properties

Vitamin / Provitamin 1)

METABOLISM2)

Active metabolite: physiological function

Deficiency disease or symptoms

Toxicity disease or symptoms

Sources

Vitamin A Provitamin: ß-carotene Vitamin: retinol

Transported

into the Lymph as retinyl esters; bound in the Blood to retinol-binding protein and prealbumin

11-cis-Retinal — component of rhodopsin and other light-absorbing pigments. Unidentified metabolites (retinoic acid?) are required for the growth and differentiation of epithelial, neural, and Bone Tissues

Children: impaired dark adaptation, Skin dryness, keratomalacia, growth retardation, death

Adults: night blindness, xeroderma

Hypervitaminosis A: headache, dizziness,

nausea, skin desquamation, bone pain

Brightly colored

vegetables (containing

carotenes),

hard margarine

Vitamin D

Provitamins: ergosterol (plants, Yeast) and 7-dehydrocholesterol (skin)

Provitamins are converted into vitamins upon ultraviolet irradiation. The vitamins are hydroxylated in the Liver to form 25-hydroxyvitamin D and in the Kidneys to form 1,25-dihydroxyvitamin D and other metabolites

1,25-dihydroxyvitamin D — the primary hormonal regulator of Mineral Metabolism (Calcium and phosphorus) in bones

Children: Rickets

Adults: Osteomalacia

Hypervitaminosis D: hypercalcemia, hypercalciuria, nephrocalcinosis

Fortified milk, sun exposure of the skin

Vitamins D2 (ergocalciferol) and D3 (cholecalciferol)






Vitamin E

Tocopherols, tocotrienols

Incompletely understood

Active metabolite unknown.

Functions as an antioxidant

Children: anemia in premature infants

Adults: no known syndromes

Not established. High doses impair Vision (blurred vision) and cause headaches

The primary source is vegetable oils

Vitamin K: K1

(phylloquinone), K2 (menaquinone), others

Incompletely understood

Active metabolite unknown, but likely a hydroquinone derivative. Activates blood clotting factors II, VII, IX, and X via y-carboxylation of glutamic acid residues; also carboxylates bone and Kidney Proteins

Children: hemorrhagic disease of the newborn

Adults: impaired Blood Coagulation. Deficiency symptoms may be induced by coumarin anticoagulants and antibiotic therapy

May be caused by Water-dispersed analogues: hemolytic anemia, liver damage

Synthesized by intestinal Bacteria

1) Fat-soluble vitamins are insoluble in water but soluble in fats and oils. They are relatively stable at ordinary cooking temperatures, but are inactivated by ultraviolet light and oxidation.

2) Dietary fat and Bile are required for the absorption of fat-soluble vitamins; malabsorption or biliary obstruction leads to vitamin deficiency. Lipoproteins or specific carrier proteins are involved in vitamin transport. Fat-soluble vitamins are stored primarily in The Liver and, to some extent, in adipose tissue. Vitamins are excreted into the bile and either reabsorbed via enterohepatic Circulation or excreted in the feces. Some metabolites may be excreted in the urine.

3) Dietary sources for all fat-soluble vitamins include green leafy vegetables, vegetable oils, fatty meats, and dairy products, In addition to the specific sources listed in the table.

Table 53.7. Vitamin-responsive syndromes. Examples of specific Metabolic Disorders of vitamin Cofactors that respond to high-dose vitamin therapy. (From: Herman R. Н., Stifel F. В., Green H. L. Vitamin-deficient states and other related diseases. In: Disorders of the Gastrointestinal Tract; Disorders of the Liver; Nutritional Disorders. Dietschy J. M. (editor). Grune and Stratton, 1976.)

Vitamin

Disease

Biochemical defect

Biotin

Propionic acidemia

Propionyl-CoA carboxylase

Vitamin B12

Methylmalonic aciduria

Cobamide coenzyme formation

Folic acid

Folate malabsorption

Folic acid transport

Niacin

Hartnup disease

Tryptophan transport

Pyridoxine (vitamin B6)

Neonatal seizures

Cystathioninuria

Homocystinuria

Glutamate decarboxylase (?)

Cystathionase

Cystathionine synthase

Thiamine

Hyperalaninemia

Thiamine-responsive lactic acidosis

Pyruvate decarboxylase

Hepatic pyruvate carboxylase



Last update: 06/08/2026

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